To those who have who have had the surgery and those who have been effectively castrated chemically:

Has any male pattern balding you had prior to castration been reversed or arrested/stopped? I have a thinning/borderline bald spot on the crown. Since I was recently gelded in March, it’s probably too soon to notice any effects regarding my head hair. I’ll be happy if the balding has at least been arrested.

Any personal experiences shared would be most appreciated.

Thanks in advance,

Evolve

Hey @Evolve01,

I'm not castrated but, I've been on Finasteride for about 17 years.

Male pattern baldness is caused by DHT (dihydrotestosterone).

DHT is created inside the body from testosterone. di means two and hydro means water. So, basically, it's testosterone with two water molecules attached to it. In other words, it's crystallized testosterone.

DHT also causes enlarged prostate - which is the reason I started taking Finasteride. Finasteride is a DHT blocker - it stops tesosterone from turning into DHT.

If you are on testosterone replacement thereapy, you will still produce DHT. If you are not on testosterone replacement thereapy, you will stop making DHT and hair regrowth or prevention of further hair loss would be equal the the way you'd respond to a DHT blocker such as Finesteride.

If you decide to go on testosterone replacement, ask your doctor about Finasteride. It is sold in both 5mg and 1mg strengths. While on patient, the 1mg was sold as a hair loss/regrowth treatment and the 5mg was sold as a treatment to shrink an enlarged prostate.

Prostate enlargement is a correct term but, most people miss exactly how the prostate is enlarging. The prostate doesn't blow up like a ballon. Instead the tissue inside the prostate grows thicker and thicker. This can squeeze the ureatha - which passes through the prostate - and make urination difficult.

A patient complaining of the symptoms of BPH (enlarged prostate) such as needing to get up more than once a night to pee, feeling the need to pee when laying down and the feeling goes away when you stand up, difficulty starting the flow of urine, a week stream of urine, feeling that you are not empty after you do go pee has all the symptoms of an enlarged prostate and would most likely be offered a DHT blocker as one of two options to treat the problem. A DHT blocker will actaully shrink the prostate tissue back down. It has the added benefit of also regrowing hair.

TMFL

Hydro is Hydrogenium. If it was water it would be "hydrat".

I was castrated about five years ago and only briefly on TRT. Prior to my orchi I had a slightly receding hairline as well as slight thinning on the crown. Both have stopped. As stated above, DHT is the culprit in male pattern baldness and once removed hair loss ceases. However, I'm not sure there would be any growth to fill in the thinning spots, but to be honest I haven't even looked.

I guess if you want to reduce drastically DHT you can switch from testosterone shots to a using Nandrolone Decanoate instead.

It's fairly simple as you take like for like dosage but reading texts on the internet you need a certain amount of DHT for health reasons.

I have heard some folks doing a 50 - 50 split between Testosterone Cypionate and ND to offset this.

I am currently just doing ND at the moment and am fine but plan to restart T in a month or so when the Vial I am using runs out.

Naturally, I am suffering a bit (Lot) with Deca Dick but I think the hair, on the top of my head, is growing longer and a bit thicker.

However, body hair seems to be a bit less but this was thinning a bit anyway.

The best scientific information that I have been able to find on the relationship between castration and baldness is a 1960 article by James Hamilton:

Hamilton, James B. (1960). Effect of castration in adolescent and young adult males upon further changes in the proportions of bare and hairy scalp. Journal of Clinical Endocrinology and Metabolism 20 (10): 1309-1318.

In the article, Hamilton reports on 21 study subjects who were castrated at ages from 15 to 28, each of whom was studied for a minimum of 8 years after castration.

ABSTRACT

A study of 21 adolescent and young adult males, before castration and for eight to eighteen years afterwards, showed that after orchiectomy there was no development of male pattern baldness (MPB) nor of any grossly recognizable denudation of the scalp. There was no expansion of bald areas in existence at the time of castration. At the end of the study the eunuchs, compared with intact males of similar age, exhibited a significantly lower incidence of MPB (P = .O1) and had no further loss of coarse hairs in the pattern that in most males results in recession of the frontal hairline (P <.O1). After castration, no increase in the number of coarse hairs was detected in bald or sparsely-haired areas of recession of the frontal hairline. It is concluded that the remedial value of drastic reduction in androgenic stimulation is probably nil with regard to return of coarse hairs which have been lost along the frontal hairline in young men. In 3 men with baldness of the crown of the head at the time of orchiectomy, a limited increase in the number of coarse hairs occurred after the operation in 1 but not in the others. Further study is required to ascertain the potential for partial regrowth of coarse hairs in subjects with late-appearing forms of MPB involving the dorsum of the head.

JesusA (imported) wrote: Fri Oct 02, 2020 9:35 am The best scientific information that I have been able to find on the relationship between castration and baldness is a 1960 article by James Hamilton:

Hamilton, James B. (1960). Effect of castration in adolescent and young adult males upon further changes in the proportions of bare and hairy scalp. Journal of Clinical Endocrinology and Metabolism 20 (10): 1309-1318.

In the article, Hamilton reports on 21 study subjects who were castrated at ages from 15 to 28, each of whom was studied for a minimum of 8 years after castration.

ABSTRACT

A study of 21 adolescent and young adult males, before castration and for eight to eighteen years afterwards, showed that after orchiectomy there was no development of male pattern baldness (MPB) nor of any grossly recognizable denudation of the scalp. There was no expansion of bald areas in existence at the time of castration. At the end of the study the eunuchs, compared with intact males of similar age, exhibited a significantly lower incidence of MPB (P = .O1) and had no further loss of coarse hairs in the pattern that in most males results in recession of the frontal hairline (P <.O1). After castration, no increase in the number of coarse hairs was detected in bald or sparsely-haired areas of recession of the frontal hairline. It is concluded that the remedial value of drastic reduction in androgenic stimulation is probably nil with regard to return of coarse hairs which have been lost along the frontal hairline in young men. In 3 men with baldness of the crown of the head at the time of orchiectomy, a limited increase in the number of coarse hairs occurred after the operation in 1 but not in the others. Further study is required to ascertain the potential for partial regrowth of coarse hairs in subjects with late-appearing forms of MPB involving the dorsum of the head.

This report gives me half a stiffy in my shorts and a shiver down my spine at man's inhumanity to man.

I'd point fingers at the doctors that conducted this experiment if it weren't for the wet spot in my underpants at reading it.

And to think I was always told the MPB was based on your genes.

gandalf (imported) wrote: Fri Oct 02, 2020 4:19 pm And to think I was always told the MPB was based on your genes.

Probably not in your genes, but maybe in your jeans.

TopManFL (imported) wrote: Thu Oct 01, 2020 5:22 am Hey @Evolve01,

I'm not castrated but, I've been on Finasteride for about 17 years.

Male pattern baldness is caused by DHT (dihydrotestosterone).

DHT is created inside the body from testosterone. di means two and hydro means water. So, basically, it's testosterone with two water molecules attached to it. In other words, it's crystallized testosterone.

DHT also causes enlarged prostate - which is the reason I started taking Finasteride. Finasteride is a DHT blocker - it stops tesosterone from turning into DHT.

If you are on testosterone replacement thereapy, you will still produce DHT. If you are not on testosterone replacement thereapy, you will stop making DHT and hair regrowth or prevention of further hair loss would be equal the the way you'd respond to a DHT blocker such as Finesteride.

If you decide to go on testosterone replacement, ask your doctor about Finasteride. It is sold in both 5mg and 1mg strengths. While on patient, the 1mg was sold as a hair loss/regrowth treatment and the 5mg was sold as a treatment to shrink an enlarged prostate.

Prostate enlargement is a correct term but, most people miss exactly how the prostate is enlarging. The prostate doesn't blow up like a ballon. Instead the tissue inside the prostate grows thicker and thicker. This can squeeze the ureatha - which passes through the prostate - and make urination difficult.

A patient complaining of the symptoms of BPH (enlarged prostate) such as needing to get up more than once a night to pee, feeling the need to pee when laying down and the feeling goes away when you stand up, difficulty starting the flow of urine, a week stream of urine, feeling that you are not empty after you do go pee has all the symptoms of an enlarged prostate and would most likely be offered a DHT blocker as one of two options to treat the problem. A DHT blocker will actaully shrink the prostate tissue back down. It has the added benefit of also regrowing hair.

TMFL

Thanks for the valuable insight about Finestrride. Given that I’m nutless and on estradiol, I probably don’t need Finestrride, but this is definitely good info for those going the chemical route or just starting out. Thanks again for sharing.