Text from above link:
WHY WAS I CASTRATED AS A 10 YEAR OLD
WITH EXPOSURE TO HUMAN GROWTH HORMONE?
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Castration.......
Performed before puberty, the male does not develop normally since the production of male sex hormone is prevented when FSH is expressed to 1.8
Concerns elvolve around
1) Secondry cryptorchidism
2) Early onset Prostate disease
3) Late onset hypospadias
4) Continuing Hypogonadism
5) Growth stunting (because God made "us" that way)
6) Premature aging
2.10 The FSH Subcommittee was established to advise on the distribution of FSH. The fertility hormone produced by CSL was not pure FSH - it also contained luteinising hormone (LH). After 1979 it was labelled as hPG and is referred to as hPG in this report. The FSH Subcommittee's official functions were to advise HPAC...
PRIVATE MEMBERS’ BUSINESS
Notices given for Tuesday, 3 June 2008
*1 FRAN BAILEY: To move—That the House:
(1) recognises the unapproved recipients of hormone treatments, including young men and boys who received human growth hormone, between 1960 and the mid 1980s;
(2) acknowledges that the report it commissioned in 1993, known as the Allars Inquiry, found that approved female patients receiving the same treatment for infertility suffered negative effects and as a result of that report, received compensation from the Commonwealth; and
(3) recognises the male recipients—both approved and unapproved—who received the same hormone treatment for growth purposes and provides similar compensation. (Notice given 3 June 2008.)
http://www.openaustralia.org/debates/?i ... er%3A10021
T
quietguy (imported) wrote: Fri Dec 05, 2008 2:59 am
he Australian Human Pituitary Hormone Program,
cut red tape, carried out illegal practises and Allars served it up in her inquiry.The program was described in the Journal Paper as an experiment and those hormones were determined (Burger et. al.) as "damaged"
These hormones were imported into Melbourne from Baltimore
HISTORICAL PERSPECTIVES (1965-1975)
In 1965, a Ross research conference on hGH was held
at The Johns Hopkins Hospital, Baltimore, Maryland.
The proceedings5 summarized the state of knowledge at
the time, including that in 1962 a radioimmune assay for
hGH was published,6 which permitted insight into GH’s
action in relation to diagnosis and treatment. By 1966,
Alfred Wilhelmi, PhD; Robert Ryan, MD, Mayo Clinic; and
Brij Saxena, PhD, Cornell University Medical College,
were extracting and purifying TSH, ACTH, LH, and FSH
from pituitaries. This ultimately permitted immunoassays
for each of these hormones to be developed. It was
possible, therefore, to significantly extend investigation
of normal and abnormal endocrine physiology
http://www.gghjournal.com/volume21/4/pd ... rticle.pdf
Treatment with FSH and LH requires approval by the Health Dept as legislated in 1965. Yet the material of LH was proven TO BE AN EXPERIMENT, with the product described as "damaged".AS EARLY AS 1968 .
http://www.nature.com/icb/journal/v46/n ... 68161a.pdf
No consent from Parents or approval from the Commissioner for Health was ever obtained by treating practioners, for those who were "unapproved"
With more than 500 "unapproved" males exposed to Human Pituitary Gonadotrophin sourced from cadavers and imported into Australia in the early 1970's, us recipiants are known as the "Unapproved" reffered to the in Allars Report. These boys treated at Prince Henry's Hospital in Melbourne were exposed to Human Growth Hormone, recorded, elevated FSH and castrated.
To date, there has only been 1 death of CJD after diagnistic infusion of hGH
Creutzfeldt-Jakob disease 38 years after diagnostic use of human growth hormone. (Short Report).(Brief Article)
A 47 year old man is described who developed pathology proven Creutzfeldt-Jakob disease (CJD) 38 years after receiving a low dose of human derived growth hormone (hGH) as part of a diagnostic procedure. The patient presented with a cerebellar syndrome, which is compatible with iatrogenic CJD. This is the longest incubation period described so far for iatrogenic CJD. Furthermore, this is the first report of CJD after diagnostic use of hGH. Since the patient was one of the first in the world to receive hGH, other cases of iatrogenic CJD can be expected in the coming years.
http://www.accessmylibrary.com/coms2/su ... 511720_ITM
Mr Smith said that the project was completed about the end of August 1993 at which time the CDs (and presumably the paper files) would have been returned to the Department of Health. Although Mr Smith did not know what the prefix "FSH" before the box number on this cover sheet referred to it is a reasonable inference that it was in fact a reference to "follicle stimulating hormone", an alternative name for HPG . Mrs Farrell's file was merely one of a large number in that series.
http://www.austlii.edu.au/cgi-bin/sinod ... ?query=hpg
"Your treatment was not to induce puberty, nor to encourage growth" according to Dr H. Teede
Reference - The Testes - Clinical and Experimental Studies, Burger Henry, de Kretser David - 1983
The medical consequences of long term castration
http://jcem.endojournals.org/cgi/conten ... 84/12/4324
Hopefully, it will never again be possible to repeat the studies reviewed in this paper, (they obviously dont know what was done to boys in Melbourne in the 1970's) as in more recent times we have used different means of expressing man's inhumanity to man. It is to the credit of the pioneering physician scientists involved that useful medical information was obtained about the long term effects of castration, under circumstances that must have been difficult, from the study of these now extinct groups of castrated men, and it is impressive that all their findings (osteoporosis, failure of closure of the epiphyses, reactive pituitary hyperplasia, shrinkage of the prostate, and development of gynecomastia) have been confirmed subsequently by studies of individuals or small groups of individuals with various forms of hypogonadism
quietguy (imported) wrote: Fri Dec 05, 2008 2:59 am
In Australia, from the early 1970's, prepubertal and adolescent boys with a prediction of 'short stature' (recruitment article, Melbourne Herald, November 1971) were exposed to hormones (human pituitary gonadotrophin, h
GH, which elevated FSH)
quietguy (imported) wrote: Fri Dec 05, 2008 2:59 am
and then treated with synthetic androgens to treat the damage, after bilateral orchidopexy
*. The batches of Hman Growth Hormone were recorded, including the elevated FSH
quietguy (imported) wrote: Fri Dec 05, 2008 2:59 am
Since then,Medicare claims between 1988 and 1994, showed 218 boys had their testes removed and 98 had vasectomies under nine years of age with a further 282 and 91 teenagers having had similar procedures aged between the ages of 10 and 19. (Carlson, Taylor and Wilson, 2000)
Yet there would be few medical reasons, if any, as to why these
procedures would happen to any male of this age. These boys and teenagers were nor hyperstimulated with pituitary hormones, but still end up with hypogonadism through castration.
"A world in which government cannot be bothered to investigate potential illegal medical assault on its citizens, in which those with no authority feel free to make decisions which are blatantly against the law and to carry out serious and irreversible procedures on those with little or no capacity to give or withhold consent, is a world in which people with disabilities can have no certainty or confidence about their human being or their future.".....Elizabeth Hastings in her statement as Federal Disability Discrimination Commissioner in 1998
The Committee also heard that in addition to the 2000 hormone recipients on the official program, possibly another 500-600 unofficial recipients were treated with "leftover" product and in other experimental programs.
http://www.bioethics.org.au/docs/Opinio ... nto%20trea tment%20of%20hormone%20recipients%20hears%20neglig ence%20allegations.doc
The Senate Inquiry (The Allars Report) failed to disclose the "unapproved recipiants" treated with Human Growth Hormone at Prince Henry's Hospital as the journal papers was never tendered. Patients continue to have difficulties in obtaining their hospital records.
These hormones were impoted from the USA and England and explains the experience of exposing males to Human Growth Hormone.
Changes in the pituitary-testicular system with age.
Clin Endocrinol (Oxf). 1976 Jul;5(4):349-72
Baker HW, Burger HG, de Kretser DM et. al.
Furthermore another 85 recipiants from Prince Henry's were not disclosed to Allars, and product was "prooven" to be damaged in a document experiment. The program was described in the Journal Paper as an experiment and those hormones were determined as "damaged"
http://www.nature.com/icb/journal/v46/n ... 68161a.pdf
Heres another 25 recipiants that were never disclosed to Allars, out of Prince Henry's
http://www.pubmedcentral.nih.gov/articl ... tid=436562
In THE AGE, Prof de Kretser stated, "were fit and well as far as I'm aware".
http://www.theage.com.au/articles/2006/ ... l?from=rss
Concerns about long term effects of these chemical interventions
have been raised, especially when drugs are given to boys whose
bone or testicular development is not complete, because of
disturbance of protein, calcium and phosphorous metabolism. Also
some men with disability may not be able to report the physical and
chemical side effects that are happening to them following the use of
these drugs, leaving them vulnerable to ill health.
There has never been any follow up to these boys who were castrated at Prince Henry's, and certainly aren't fit nor well!. and no studies have elvolved into being an invivo experiment with cadavers brain organs.
[1] Human pituitary gonadotropin is sometimes abbreviated to hPG and sometimes referred to as follicle stimulating hormone or FSH or human pituitary follicle stimulating hormone or HPFSH. Farrell v CSL Limited & Anor [2004] VSC 308 (26 August 2004).
Records showed "overtreatment" shown as FSH (12 April 1972)
9.108 (Allars pg. 620) In 1985 a nurse of some of the treating medical practitioners at Prince Henry"s, forwarded to the Deptartment of Health a list of hPG recipients in response to the third Department letter. One of the treating medical practitioners did not recall this request of the Department in 1985.
Clinicians noted in their current study that, when cryptorchidism was self reported in either the fertile or infertile population, it was accompanied by lower sperm counts, lower serum inhibin B concentrations (a marker of Sertoli cell function), and higher LH and lower testosterone to LH ratios. They propose that these data support the possibility that impaired androgen production may be an additional marker of TDS.
J Clin Endocrinol Metab, July 2004, 89(7):3158–3160
D. M. de Kretser
Endocrine evaluation Although gonadotropin deficiency is uncommon, diagnosis of hypogonadotropic hypogonadism is important since treatment is available. The measurement of serum follicle-stimulating hormone (FSH) is generally regarded as valuable to distinguish patients with azoospermia who have obstruction (normal FSH) from those who have seminiferous tubule destruction (raised FSH).
http://www.mindfully.org/Health/Pathoge ... tility.htm
And hence....oxandrolone (testosterone) was used to enduce puberty, but not according to Teede H.
Androgen physiology
Testosterone is the predominant androgen in males with 95% being secreted by testicular Leydig cells under the influence of luteinising hormone (LH) from the pituitary gland.
Australian Family Physician Vol. 32, No. 6, June 2003
(ii) At the same time HREOC worked directly with the Commonwealth Department of Health & Aged Care for changes to the Medicare Benefit Schedule. On 1 November 1998 the "Notes for Guidance" in the Medicare Benefit Schedule were amended to include the following guidelines:
"It is unlawful throughout Australia to conduct a sterilisation procedure on a minor (under 18 years of age) which is not a by-product of surgery appropriately carried out to treat malfunction or disease (eg; malignancies of the reproductive tract). Parents and guardians have no legal authority to consent on behalf of minors to such sterilisation procedures. Since when is being "short" a disease? - because God made us that way?
No consent was obtained by practitioners from Parents to have their sons castrated.
b) Procedures which go beyond sterilisation
(i) Males
2.5 A method of male sterilisation performed from antiquity is castration, being the removal of the testes or male sex organs themselves.5 However, this procedure goes beyond sterilisation to affect the sexuality of the male concerned. Performed before puberty, the male does not develop normally since the production of male sex hormone is prevented. Performed after puberty, secondary sexual characteristics may be affected in tha t the male may cease to grow a beard and may develop breasts. Since the voices of castrated males did not deepen at puberty, in Italy until the 20th century certain male children were castrated in order to provide singers for the 'castrati' parts in Italian opera, and for the Vatican and other church choirs. Oriental rulers employed eunuchs, castrated males, as keepers of their harems.
Final Report on Consent to Sterilisation of Minors (1994)
According to Lord Denning MR in King v Victor Parsons & Co [1973] 1 All ER 207 at 209 - 210 it would be 'against conscience' for a Defendant to avail himself of the lapse of time if he took positive steps to keep a plaintiff ignorant of his or her right of action after it has arisen. If a man knowingly commits a wrong in such circumstances that it is unlikely to be found out for many a long day, he cannot rely on the Statute of Limitations as a bar to the claim:. and
2. According to Lord Denning In order to show that he 'concealed' the right of action 'by fraud', it is not necessary to show that he took active steps to conceal his wrongdoing or his breach of contract. It is sufficient that he knowingly committed it and did not tell the owner anything about it. (The Melbourne boys were never disclosed to the Senate Inquiry, nor the Journal Papers where pituitary hormones were imported). He did the wrong or committed the breach secretly. By saying nothing he keeps it secret. He conceals the right of action. He conceals it by 'fraud' as those words have been interpreted in the cases. To this word 'knowingly' there must be added 'recklessly'
Like the man who turns a blind eye. He is aware that what he is doing may well be a wrong, or a breach of contract, but he takes the risk of it being so. He refrains from further enquiry lest it should prove to be correct; and says nothing about it. The court will not allow him to get away with conduct of that kind. It may be that he has no dishonest motive; but that does not matter. He has kept the plaintiff out of the knowledge of his right of action; and that is enough: see Kitchen v Royal Air Forces Association [1958] 2 All ER 241, [1958] 1 WLR 563. If the defendant was, however, quite unaware that he was committing a wrong or a breach of contract, it would be different. So if, by an honest blunder, he unwittingly commits a wrong (by digging another's man's coal), or a breach of contract (by putting in an insufficient foundation) then he could avail himself of the Statute of Limitations."
Practitioners may be subject to criminal and civil liability action if the sterilisation procedure is not authorised by the Family Court of Australia or a Court or tribunal with jurisdiction to give such authorisation".
http://www.hreoc.gov.au/disability_righ ... chap5.html
For a plea of concealment by fraud to succeed on the basis of non-disclosure, it is necessary to plead and prove that the individuals responsible for the non-disclosure were conscious that such non-disclosure was wrongful, or should have been conscious that it was wrongful. In Seymour v Seymour (supra) at 372, Mahoney A-CJ, with whom Meagher JA and Abadee A-JA agreed, said the following in relation to the New South Wales equivalent of s32(1)(b):
"In my opinion, the section is not confined to simple common law fraud. It extends to conduct beyond that. On the other hand, it is not, I think, sufficient merely that for the defendant to take advantage of the statute of limitations would be unconscionable or inequitable in the wide sense of these terms. Terms such as unconscionable and inequitable now are used to describe conduct which, in previous times, would not have fallen within them: see Baumgartner v Baumgartner (1987) 164 CLR 137 at 147 and Hibberson v George (1989) 12 Fam LR 725 at 731.
Nor, in my opinion, is 'fraudulently' wide enough to include everything which would fall within the description of 'equitable fraud'. Equitable fraud is a doctrine which depends, for this purpose, too much upon nice distinctions which have been drawn in other times: see Snell's Equity (29th ed) at 550 et seq; Meagher, Gummow & Lehane (3rd ed) at par 1208; and see the discussion in Logue v Shoalhaven Shire Council [1979] 1 NSWLR 537 at 553. The history of the English legislation was recently reviewed in Sheldon v RHM Outhwaite [1996] 1 AC 102: see, eg, at 144, 153.
In my opinion, there must be in what is involved a consciousness that what is being done is wrong or that to take advantage of the relevant situation involves wrongdoing. At least, this is so in the generality of cases. (There is in this as in many things, the problem of dealing with the person who 'closes his eyes to wrong' or is so lacking in conscience that he is not conscious of his own lack of proper standards.)"
It was known as early as 1966 that castration was achieved in animal studies using pituitary hormones.
The Senate Inquiry failed to make comment and refrained to make recommendations, as to having the Practitioners charged with criminal offences. In France (and elsewhere) practioners were charged, convicted and jailed. In Australia, the boys are only able to access their medical files through the courts ; the hidden agenda is not to disclose (castration) to the recipients.
The Family Law Council recommended four circumstances in which sterilisation should never be authorised. These are:
Sterilisation for eugenic reasons;
Sterilisation purely for contraceptive purposes;
Sterilisation as a means of masking or avoiding the consequences of sexual abuse;
"It is also important to acknowledge the participation of numerous patients in the studies described. Without their help, clinical research would not be possible and quite often their misfortune prompted a new study or helped to elucidate a novel concept"
The Pituitary and Testes - Clinical and Experimental Studies 1983. Burger HG, de Krester DM
Apart from medical records being kept hidden from patients, certain journal documents were never presented to The Allars Inquiry pertaining to the use of pituitary hormones used at Prince Henry's Hospital. The clinicians kept their experiements hidden from the Allars Inquiry.
The boys were hyperstimulated effecting the testes (castration), and went onto have corrective surgery to replace the ectopic testes back into the scrotum after being hyperstimulated.
Between 1967 and 1985 cadaver derived pituitary hormones were officially supplied to 1,976 Australians as treatment for infertility and short stature. In addition, 187 people are known to have been treated with human pituitary derived hormones in Australia as part of special projects associated with metabolic growth problems, and between 1972 and 1978 on some IVF programs. A number of people also received treatment outside these programs, as early as 1963. A total of 2,163 people were treated with human pituitary derived hormones in Australia during this time.
It is unknown the full extent of males treated with by the chairperson of one of the subcommittees of the AHPHP, including boys exposed to Human Growth Hormone at Prince Henry's Hospital are included in these figures; however in submission to the Senate Affairs reference Committee details emerged of 500-600 "unapproved" recipients of pituitary hormones. These "unappoved" have never been officially notified of their risk of CJD, from hGH.
Submitted to the Senate Affairs Reference Committee (1997), CSL produced a document to treat males with hPG and warned of the hyperstimulation effects. CSL also recorded the effects of hypogonadism with hyperstimulation with hPG.
Like the "Tall Girls" who were treated with diethylstilbestrol (DES, a sex steroid of estrogen) to "stunt" their growth, boys were treated with Oxandrolone (a sex steroid of testosterone) which also "stunted" growth after a "growth spurt".
I went onto have corrective surgery to replace the ectopic testes back into the scrotum.
WARNING - THIS SITE ADRESSES THE ISSUES OF HORMONE DISTRUPTION UPON PREPUBERTAL MALES WITH DEVISTATING SIDE SIDE EFFECTS INCLUDING HYPOGLYCEMIA,CASTRATION, CANCERS AND EVEN DEATH.
Hypogonadism (i.e. oxandrolone induced, as a growth promotant), according to Prof Connell is also a fatal disease.
Its the "unapproved" group who were treated with pituitary hormones prepubertally, overtreating the testes, causing castration, enduring bilateral orchidopexy for ectopic testes, then treated with testosterone due to damaged testes that inhibited the secretion of sex steroids.
In submission to the Senate Affairs Reference Committee (1997), CSL produced documents with guidelines for males to be treated with hPG/FSH who suffered from hypogonadism. The FSH Subcommittee called it the LH Program with CSL supplying FSH/hPG for male hypogonadism. These hormones (pituitary derived) were imported from the USA and England.
Hypogonadism in childhood is "normal" for boys, and as such cannot be diagnosed until the age of 16-17 years. Exposing prepubertal males to a "hormone blocker" had devistating side effects.
While androgen deficiency (testicular damage) may not shorten life expectancy, (although now questionable by Prof Connell) its pervasive effects on the development and maintenance of masculine sexual characteristics and anabolic status of somatic tissues can impoverish quality of life. (Zajac, 2004).
However in evidence in Queensland in August 2005, according to Prof Connell, Hypogonadism is also a fatal disease.
Hyperkinetic Syndrome (aka ADHD), during and post oxandrolone treatment, (hyperkinetc syndrome) was treated 30 years ago with beta blockers as stimulants werent widely available. Apart from Oxandrolone as treatment for the hyperstimulation of FSH for damage to the testes (hypogonadism), Inderal and Tofranil were the early treatments available for the Hyperkinetic sydrome.
The orthostatic tachycardia syndrome is a disabling disease state described at least since 1940 and is the most common reason for referral for chronic orthostatic intolerance. Patients are often unable to hold jobs or attend schools.
Since the 1970s the general mode of treatment of ADD/HD has been the use of psychostimulate drugs such as Ritalin (methylphenidate), Cylert (pemoline), Dexedrine (dextroamphetamine) and Adderall. In addition antidepressants such as Norpramin (desipramine), Tofranil (imipramine), Wellbutrin (buproprion), BuSpar (buspirone), Prozac (fluoxetine), Corgard (nadolol), Inderal (propanol) and Clonidine have been prescribed. Most scientists believe that these drugs simulate the production of neurotransmitters in the brain. There is no evidence to indicate any permanent improvement in the patient’s symptoms. In addition, once the patient stops taking these psychostimulant drugs, any positive effects it had disappears. Orthostatic Hypotension is treated with Inderal.
Animal studies in the mid 1960's showed young male calves exposed to Pituitary Hormones were made cryptorchid (Senate Submission), and experiments on the boys had the same effects in the early 1970's, yet even with this data the doctors involved went ahead with the blessing of the Federal Government, and no one has ever been made accountable. The Federal Government continues to protect these practitioners who (according to the senate Inquiry) were acting illegally.
And in 1972 more information........Inhibin is a glycoprotein hormone, which inhibits the production and/or secretion of the pituitary gonadotropins, preferentially FSH. Immunoreactive inhibin levels were elevated at the time of hypothalamo-pituitary-testicular axis activation in young boys and rose during normal human puberty, being correlated with rising levels of the gonadotropins and testosterone.
http://endo.endojournals.org/cgi/conten ... 138/4/1361
Some side effects for myself, to the Program including Traumatic Brain Injury with vestibular dysfunction of the cranial nerve, Post Adreanal Insifficiency leading to hospitalization, including salt wasting, with volume depletion is the hallmark of this syndrome. Clinically, patients manifesting CSW are dehydrated, lose weight, (hypoglycemia, hyperpigmentation, low blood pressure, sugar and/or salt cravings, anorexia, CFS) have orthostatic hypotension, (leading to a nervous breakdown) and demonstrate a negative fluid balance.
Adrenal insifficiency include progressive weakness, fatigue, anorexia, nausea & vomiting, weight loss, hypotension, hyperpigmentation in skin and oral mucosa due to >ACTH and POMC precursor
Notwithstanding attempted Suicides ( a withdrawal effect to steroids)and Ideation.
The oxandrolone was used in the belief to accelerate linear height, but oxandrolone (testosterone) was used for pubertal hypogonadism (damaged testes), after hyperstimulation of the gonads, in which required an operation (bilateral orchidopexy) to place them back into the scrotum.
Synthetic steroids acts as a hormone blocker, and Tanner Stage 5 puberty was never achieved. Oxandrolone is no longer used as a growth promotant, due to side effects.
The "funded" agenda, and the short statured boys werent advised was the secret development of the male birth control pill in collaboration with the World Health Organization (WHO) (refer to website). (1974)
"Three decades on, the male pill has still not arrived. The most recent breakthrough came earlier this month (October 2003), with the announcement by Australian scientists of a treatment involving an implant under the skin — meaning men could not forget to take it — of hormones that switch off sperm production. It’s said to be 100 per cent effective and free from unpleasant side effects."
FSH levels rose 9 fold after "overtreated" with Human Growth Hormone (precosious puberty - neuroendocrine with hPG and doubling testosterone levels, and raising FSH levels. Bilateral orchidopexy was required to relocate the ectopic (perineum and ingual) testes back into the scotum 6 weeks after exposure to Human Growth Hormone.
The infusion upon the testes as indicated with an elevated FSH level and testosterone increase from 122 to 250 ng.
Elevated FSH levels are seen when the testis (and spermatogenesis) has been damaged (primary testicu-lar failure). Its clinical term is Primary Hypogonadism.
7.56 . It also developed additional assays requested by HPAC, for example the luteinizing hormone assay, introduced in 1972.[71].
The Senate Inquiry failed to record the effects of overtreatment upon males (ectopic testes)
One book derived from PHH describes the death of a boy on the program.
Never disclosed to the Inquiry was the hyperstimulation effect on the boys at Prince Henry's, and the short term and long term effects upon the testes. Cryptorchidism (ectopic testes) was certainly a side effect to the exposure of Human Growth Hormone, an experiment never disclosed to the boys, nor concented to.
Hyperpigmentation (adrenal dysfunction), Melanoma, Basal Cell Carcinoma, nocturnal enuresis, prostate disease (as a teenager), gynecomastia, IBS, fluid retention, inreased bladder muscle markings (neuogenic bladder), knock knee (Paget's disease), heart disease, vestibular dysfunction (cerebellar ataxia) associated with , Myoclonic jerks, muscle spazms, Low FSH and Testosterone, Skeletal dysplasia, Liver disease, and growth stunting to name a few side effects, notwithstanding puberty was never completed, due to exposure of a hormone blocker prepubertally. Attempting suicide through self harm as a withdrawal effect, and enduring nervous breakdowns were apart of the experience of endocrine disruption with hormones and sex steroids.
Concerns are raised upon page 460 of The Allars Report where The Subcommittee approved themselves to Hyperstimulate with hPG. Disclosure and side effects of the potential deaths (of hyperstimulation) and castration wasnt concented to by the subjects, nor their Parents.
Growth Hormone treatment used in 10 year old boy that had devistating effects. Left with hypogonadrophic hypogonadism, I didn't complete Tanner Stage V Puberty, and have been refused treatment for the last 25 years. Hypogonadism is a fatal disease according to Prof Connell,and hypogonadal males have a shortened life span.
Biochemical premature aging known as Progeria (age based testosterone levels) along with prostate disease as a teenager, along with Testosterone/Estrogen imbalance without treatment for 25 years has taken its toll. Free testosterone levels were non existant.
During the 1970's, the Growth Hormone secreted me from .2 to 1.8 (the adult range in a 10 year old).
It was documented that in prepubertals, normal range was <.3 to 1.3,not 1.8
http://www.nature.com/icb/journal/v46/n ... 68161a.pdf
Oxandrolone kicked the testosterone levels to Tanner Stage 111 (14-18 year old) as a 12 year old, with FSH rising. Tanner Stage V was never achieved. Precosious Puberty, as seen with elevated FSH levels, (indicitive of castration) as a 10 year old with an adult range (libido) was most concerning.
Premature aging, through biochemical diagnosis of hormonal levels of a male aged 100, 30 years on is a major consequence of the refusal of treatment for the condition. By the mid 30's experience shows that testosterone levels where that of an 85-100 year old male and physical strength had diminished to non existant levels. Without treatment, "old aged" males are bedridden in their 40's.
It becomes a major concern when the hPG/hGH administration has been associated with deaths of CJD - the human equivilant of BSE/Mad Cow Disease, with an incubation period of upto 38 years after the Stimulation Test for GH deficiency. Pituitary hormones were imported for use at Prince Henry's Hospital and were recorded during the excersise tests.
Batches of hGH were never recorded on file notes, however the elevated FSH was also recorded.
.................................................. .................................................. ...
The problem with many of the compounds that have been used over the years was side effects.
Although it is quite simple to make a man infertile, it has to be achieved with an acceptable level of side effects.
Various compounds have had different side effects, including acne, weight gain, increased appetite, mood changes, lethargy and longterm impotence. Some of the side effects experienced are similar to those reported by women on the Pill. Unlike womens estrogen to be "replaced" every month, male testosterone levels drop off over the years, in these cases, prematurely, leading to premature aging.
"Many of the chemicals that have been tried do work, but they also have side effects," Robaire says. "You can easily block the production of the male hormone, and then you obviously have a contraceptive, but that is equivalent to a castration, and that is not good."
http://www.theage.com.au/articles/2003/ ... click=true
.................................................. .................................................. ....
2.70 In evidence Professor Whitworth stated:
The Human Pituitary Advisory Committee instituted a requirement that any specialist seeking to be approved by them to treat patients with hPG (Human Pituitary Gonadotrophins) must have access to a laboratory to carry out the assays required. A computerised recording system was developed in the [Health] department which doctors were required to provide information to on the outcome of treatment. Certainly, hyperstimulation remains a side effect of gonadotropin treatment to this present day.
Prior to 1979, (1972-1976), FSH was a combination of FSH and LH. with the importation of FSH and LH from overseas pituitary glands, a gift from the National Pituitary Agency (Bethesda, Maryland)and Dr Anne Stockell Hartree (University of Cambridge, England),
*Changes in the pituitary-testicular system with age.
Clin Endocrinol (Oxf). 1976 Jul;5(4):349-72
Baker HW, Burger HG, de Kretser DM et. al.
No thorough multidisciplinary, long-term follow-up has ever been done with regard to this treatment, according to de Kretser (The Age January 2006), the NHMRC Recinded the "follow up application".
There is however evidence about their concerns since the fact that Oxandrolone treatment was discontinued in 1990 and other Growth promotors such as Human Growth Hormone (hGH) has now ceased being manufactured from Pituiarty Glands in 1985.
Hudson of Melbourne University, was leading a United Nations' task force hoped to give the world a male birth control pill. The funding was granted by the NHMRC.
The boys at PHH were treated with oxandrolone (an anabolic steroid) that encouraged growth but prematurely closed the growth plates (12 month of ingestion), resulting in knock knee. Other concerns surround the estrogenic effects with the conversion of testosterone into estrogen, leaving myself with gynecomastia and prostate disease (BPH) as teenagers.
We hope this group will be able to support you and help with the side effects of this hormone program. The program that was an in-patient ("in vivo") experiment, without consent or "approval".
One recipient told the committee "All who conspired to force this terrible legacy on hPG and hGH [human growth hormone] recipients are now being protected by a government and its officers who would rather see innocent recipients denied justice than admit to the ineptitude and negligence of those involved in producing these treatments and administering this program."
*Incidence of testicular ascent in boys with retractile testes.Stec AA, Thomas JC, DeMarco RT, Pope JC 4th, Brock JW 3rd, Adams MC.
Division of Pediatric Urology, Vanderbilt University Medical Center, Nashville, Tennessee 37232-9820, USA.
PURPOSE: The incidence of testicular ascent of a previously descended but retractile testis is reported to be between 2% and 45%. It has been our bias that only a small percent of retractile testes ascend and ultimately require orchiopexy. MATERIALS AND METHODS: We performed a retrospective review of all retractile testes diagnosed by any of 4 pediatric urologists at our institution from 1996 to 2005. A retractile testis was defined as one that could be brought to at least the mid scrotum without tension despite a history of maldescent or a retractile nature. Patients were excluded if followup was less than 6 months or they had a history of ipsilateral inguinal surgery. A followup telephone survey was done in patients not reexamined at resolution. RESULTS: A total of 172 patients met our inclusion criteria and were followed to resolution. A total of 274 retractile testes were initially identified, including 26% unilateral and 74% bilateral testes. A total of 19 testes ascended and ultimately required orchiopexy within a followup of 6 to 101 months. A patent processus vaginalis was identified in 13 cases (68%). Another 235 patients initially seen with a diagnosis of retractile testis who had no followup examination at our institution were contacted via telephone. This set of boys initially had a total of 392 involved testes, of which 2 underwent orchiopexy elsewhere before resolution. The incidence of secondary ascent was 3.2% in the combined set of patients. CONCLUSIONS: Retractile testes can ascend and, therefore, they require followup until resolution. In our experience most retractile testes do not require surgery.
PMID: 17707014 [PubMed - indexed for MEDLINE]
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