erikboy (imported) wrote: Tue Apr 21, 2009 1:13 pm Sorry for my ignorance, but isn't it so that Androcur is blocking T receptors from T influence in your body and is not reducing T production directly? Thus, making T tests shouldn't give too relevant indication about "castration levels"?!

So T is still flowing in your veins, only it is useless to your body?

Thus T level decrease is a result of testicle athropy indirectly induced by androcur rather than stopping testicles to produce T directly?

From what I have read you are right but how does one objectively test where one is rather than just go on subjective feelings?

erikboy (imported) wrote: Tue Apr 21, 2009 1:13 pm Sorry for my ignorance, but isn't it so that Androcur is blocking T receptors from T influence in your body and is not reducing T production directly? Thus, making T tests shouldn't give too relevant indication about "castration levels"?!

So T is still flowing in your veins, only it is useless to your body?

Thus T level decrease is a result of testicle athropy indirectly induced by androcur rather than stopping testicles to produce T directly?

Thanks. Great one.

This is what my urologist at vietnam told me. This was , as he said, the most important reason why the chemo could do its job, and if I am that sensitive to anything that is going on in my body, I would still feel the presents of testosterone, and be unhappy with it.

Maybe your view is right, maybe testicle athropy, is what makes me so happy this moment.

Both, my family doctor in NL and the urologist at Vietnam didn't see the need for doing T tests according to the use of androcur. So I don't know about my levels. Or should I say, I do know, I can feel it, but I can't tell you any numbers

loveUall

Jean

unencumbered (imported) wrote: Tue Apr 21, 2009 2:16 pm From what I have read you are right but how does one objectively test where one is rather than just go on subjective feelings?

It seem to me that subjective feeling is the most appropriate measure. Unfortunately.

Could anyone who is more educated explain the mechanics how androcur makes testicles to atrophy?

There is a hormanal system that releases gonadotropins depending on T levels. But when androcur block T from every receptor this system must go crazy in attempt to increase T production? (causing hot flashes) and when this system shuts down finally, testicle atrophy and decrease of actual T levels follow. While effects of castration are felt as soon as androcur replaces T in receptors?

Am I correct?

erikboy (imported) wrote: Wed Apr 22, 2009 7:35 am It seem to me that subjective feeling is the most appropriate measure. Unfortunately.

Could anyone who is more educated explain the mechanics how androcur makes testicles to atrophy?

There is a hormanal system that releases gonadotropins depending on T levels. But when androcur block T from every receptor this system must go crazy in attempt to increase T production? (causing hot flashes) and when this system shuts down finally, testicle atrophy and decrease of actual T levels follow. While effects of castration are felt as soon as androcur replaces T in receptors?

Am I correct?

And my Dutch leaflet tells me that androcur/ cyproteroneacetate is used after surgical castration to avoid hot flushes (eng) / flashes (american)

I don't understand anything any more

loveUall

Jean

test lab is ok!!!

T level is 56 - before 470

Free T level is 4 - before 30

prolattine is 14 - before 5

do you think i'm castrated???

bie