Penis shrinkage due to zero testosterone
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tinypenis (imported)
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Penis shrinkage due to zero testosterone
I'm very curious to know how much penis shrinkage occurs with zero (or near zero) testosterone levels. Of course, with that level of testosterone, nocturnal erections no longer occur. Considering that nocturnal erections keep a man rock hard for hours every night, their absence has got to have a major effect on penis size over time.
So, if you have experience in this area, or if you have good anecdotal evidence, can you answer some or all of these questions?
1.) how much does the penis shrink with zero testosterone?
2.) how fast does it shrink?
3.) how much does the girth decrease?
4.) does it eventually stop shrinking, or does it just keep getting smaller and smaller?
5.) are there any other changes, like foreskin regrowth, penis retreating into the body, scrotum shrinkage, etc.?
So, if you have experience in this area, or if you have good anecdotal evidence, can you answer some or all of these questions?
1.) how much does the penis shrink with zero testosterone?
2.) how fast does it shrink?
3.) how much does the girth decrease?
4.) does it eventually stop shrinking, or does it just keep getting smaller and smaller?
5.) are there any other changes, like foreskin regrowth, penis retreating into the body, scrotum shrinkage, etc.?
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tugon (imported)
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Re: Penis shrinkage due to zero testosterone
As a 14 year no T eunuch the penis does shrink. Once shrunk it does not shrink anymore. Well it does when you are out in the cold walking your dog. Sadly to the point it barely sticks out of both your underwear and your jeans. Difficult to piss behind a tree and with the dog pulling at the same time. But I digress.
I would say I lost about 40% of length and 25% of girth. My scrotum has also retracted close to my body. My penis has not retreated into my body but I have noticed that with obese older men it can retreat. Knowledge from my patient care days regarding older men's penises.
Even on hot summer days I still look like I jumped into ice cold water. A visual to help you understand how things look.
I would say I lost about 40% of length and 25% of girth. My scrotum has also retracted close to my body. My penis has not retreated into my body but I have noticed that with obese older men it can retreat. Knowledge from my patient care days regarding older men's penises.
Even on hot summer days I still look like I jumped into ice cold water. A visual to help you understand how things look.
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cast1988 (imported)
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Re: Penis shrinkage due to zero testosterone
after my castration my penis shrunk about 25% but my empty scrotum is still hanging down and is the same size but it's only been about 6 months since i lost my testicles
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janekane (imported)
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Re: Penis shrinkage due to zero testosterone
I am only one of quite a few folks here who have extensive education and work experience in clinical medicine and medical research settings and who have undergraduate and graduate degrees in one or another form of biology.
That having been said, my decision, in 1986, to get my bilateral orchiectomy was motivated by my noting a number of relatives who died comparatively young (including during their fourth decade of life), and recognizing my most likely having the particular genetic condition which "caused" such cancer. 25 years later, it has become incontrovertible that I have a form of the genome for familial adenomatous polyposis (FAP); and the medical information I had in 1986 strongly suggested to me that testosterone was a contributing factor, in my particular family, to the development of terminal cancer.
The State of California includes testosterone on its list of known carcinogens. However, FAP does not develop because of carcinogens alone; the FAP gene produces a tumor suppressor protein which, in the presence of a tumor cell, apparently activates apoptosis to prevent cancer development.
While the common view as I have found it in the medical literature is that FAP is of dominant inheritance, I find it to be a recessive gene at a fragile locus, such that inheriting a non-functional tumor protein gene from one parent does not result in failure of tumor suppression until the functional allele in a cell mutates somatically into a non-functional form, such that, in a cell having two non-functional tumor suppressor protein genes, tumor suppresson (apoptosis) will cease.
Because tumor cells form frequently, everyone would plausibly die in utero from cancer absent effective apoptosis resulting from functionally effective tumor suppresor genes.
Apoptosis? Programmed cell death. Apoptosis is why people are not usually born with webbed fingers and toes. In ducks, apoptosis not working in feet results in ducks having webbed feet. Programmed cell death, with different programming, is why ducks ordinarily have webbed feet and people do not. Ducks do not usually have a functional apoptosis program for the inter-toe web; people usually do.
Or, to use the classic Eunuch Archive phrase, "your mileage may vary."
After 25 years as a "no testosterone" eunuch, it is my observation (I am rather good at making biological measurements) that I have not experienced any penis shrinkage as a result of testosterone avoidance.
Unlike many folks, or so it seems to me, my being autistic may have given me an advantage in expecting the results of my orchiectomy to be mostly what I could not otherwise expect; in expecting to find results which I was unable to expect, I was never disappointed. I expected shrinkage. Had I wanted that, surely I would be very disappointed by now.
However, I seem to have an inner way of living which is, as I experience it, profoundly and gently affirming of my life and life experiences. I anticipate being unable to accurately anticipate the details of very nearly every event in my life, and my way of adapting to life experiences is, consequently, awfully simple.
Something happens (i.e., some life event which I have not accurately anticipated) and I adapt to it (or learn from it) as I am able to adapt (or learn), and the way I am able to adapt and learn is always, in my life experience, both necessary and sufficient; and whether I adapt to an event by remaining alive or by becoming dead (adapting by becoming truly dead seems to not have really happened yet); and if I remain alive, I will experience more events to which my adaptation and learning will always, in my view, be both necessary and sufficient.
Thus, I have never learned to actually hate any aspect of my life or any aspect of any aspect of life.
Had avoiding testosterone for more than 25 years resulted in my penis shrinking, I would have adapted to that. Observing that my penis did not notably shrink resulted in my adapting to that.
However, my adaptation to life events has never become passive. I actively work away at the effort I make to adapt to my life experiences, doing so with all the effort I can muster, doing so unrelentingly.
Did my scrotum shrink? Not that I can tell. Do I want it removed? No, because I avoid surgery whenever possible because I can see no benefit to me commensurate with the very small risk a "scrotectomy" would convey.
Not autistic? The way I adapt to life experiences may not be appropriate for you. But that is not for me to say for sure, not one way nor another.
That having been said, my decision, in 1986, to get my bilateral orchiectomy was motivated by my noting a number of relatives who died comparatively young (including during their fourth decade of life), and recognizing my most likely having the particular genetic condition which "caused" such cancer. 25 years later, it has become incontrovertible that I have a form of the genome for familial adenomatous polyposis (FAP); and the medical information I had in 1986 strongly suggested to me that testosterone was a contributing factor, in my particular family, to the development of terminal cancer.
The State of California includes testosterone on its list of known carcinogens. However, FAP does not develop because of carcinogens alone; the FAP gene produces a tumor suppressor protein which, in the presence of a tumor cell, apparently activates apoptosis to prevent cancer development.
While the common view as I have found it in the medical literature is that FAP is of dominant inheritance, I find it to be a recessive gene at a fragile locus, such that inheriting a non-functional tumor protein gene from one parent does not result in failure of tumor suppression until the functional allele in a cell mutates somatically into a non-functional form, such that, in a cell having two non-functional tumor suppressor protein genes, tumor suppresson (apoptosis) will cease.
Because tumor cells form frequently, everyone would plausibly die in utero from cancer absent effective apoptosis resulting from functionally effective tumor suppresor genes.
Apoptosis? Programmed cell death. Apoptosis is why people are not usually born with webbed fingers and toes. In ducks, apoptosis not working in feet results in ducks having webbed feet. Programmed cell death, with different programming, is why ducks ordinarily have webbed feet and people do not. Ducks do not usually have a functional apoptosis program for the inter-toe web; people usually do.
Or, to use the classic Eunuch Archive phrase, "your mileage may vary."
After 25 years as a "no testosterone" eunuch, it is my observation (I am rather good at making biological measurements) that I have not experienced any penis shrinkage as a result of testosterone avoidance.
Unlike many folks, or so it seems to me, my being autistic may have given me an advantage in expecting the results of my orchiectomy to be mostly what I could not otherwise expect; in expecting to find results which I was unable to expect, I was never disappointed. I expected shrinkage. Had I wanted that, surely I would be very disappointed by now.
However, I seem to have an inner way of living which is, as I experience it, profoundly and gently affirming of my life and life experiences. I anticipate being unable to accurately anticipate the details of very nearly every event in my life, and my way of adapting to life experiences is, consequently, awfully simple.
Something happens (i.e., some life event which I have not accurately anticipated) and I adapt to it (or learn from it) as I am able to adapt (or learn), and the way I am able to adapt and learn is always, in my life experience, both necessary and sufficient; and whether I adapt to an event by remaining alive or by becoming dead (adapting by becoming truly dead seems to not have really happened yet); and if I remain alive, I will experience more events to which my adaptation and learning will always, in my view, be both necessary and sufficient.
Thus, I have never learned to actually hate any aspect of my life or any aspect of any aspect of life.
Had avoiding testosterone for more than 25 years resulted in my penis shrinking, I would have adapted to that. Observing that my penis did not notably shrink resulted in my adapting to that.
However, my adaptation to life events has never become passive. I actively work away at the effort I make to adapt to my life experiences, doing so with all the effort I can muster, doing so unrelentingly.
Did my scrotum shrink? Not that I can tell. Do I want it removed? No, because I avoid surgery whenever possible because I can see no benefit to me commensurate with the very small risk a "scrotectomy" would convey.
Not autistic? The way I adapt to life experiences may not be appropriate for you. But that is not for me to say for sure, not one way nor another.
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Hash (imported)
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Re: Penis shrinkage due to zero testosterone
I have to agree with tugon, but it's taken a few years to shrink that much. It's juvenile or pre-puberty looking to me.
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gandalf (imported)
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Re: Penis shrinkage due to zero testosterone
I have been a eunuch now for 6.5 years, have lost my scrotum (after 3 years of eunuchism). My penis has shrunk about 45 percent maybe a little less when I don't have an erection (which is hard to come by). I read somewhere that part of the shrinkage is due to little or no erections. Don't know if that is right and can't remember where I read it. All I know is my "equipment" did shrink. Not so much in girth as in length.
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littletits (imported)
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Re: Penis shrinkage due to zero testosterone
My penis shrinkage has neen dramatic to say the least. I had a fairly average penis prior to castration. It is now less than half the length it was. I can never get erect due to damage to the organ in the incident that resulted in me becomong a Eunuch and I have little sensation in it. My scotum is also very small and does not hang down betwen my legs but remains close to my body and when cold it looks as though it is no longer there. The shrinkage had all occurred in the first year to a year and a half. I have not noticed it shrinking further since. My scrotum and pubic bone suffered damage at the time and this may have caused the shrinkage to be more pronounced.
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hkeunuch (imported)
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Re: Penis shrinkage due to zero testosterone
I have been a chemical eunuch for a few years now. The penis, which was never big to start with, shrank noticeably. It started as a cut cock, and now under normal circumstances, it has a full foreskin covering the entire glans. It took me a few years to reach this state. Girth did shrink some, but not nearly as noticeable.
Now that I have been off Androcur for a couple of years already, it remains as small as before. It did not seem to get much smaller.
I have always been into stuffing my genitals into my body. Now sitting down, penis needs no help to retreat into the body. But I guess I don't have sufficient fat around to keep it in all the time.
Scrotum does not really shrink much per se. But because the testes shrank a lot (to about 1/3 of their original size, clearly smaller than the bunched up vas deferens), there is no longer enough weight or volume to keep the scrotal sac stretched / hang down any more. But the skin is still there, but it does seem to generally bunch up nearer to the body.
Now that I have been off Androcur for a couple of years already, it remains as small as before. It did not seem to get much smaller.
I have always been into stuffing my genitals into my body. Now sitting down, penis needs no help to retreat into the body. But I guess I don't have sufficient fat around to keep it in all the time.
Scrotum does not really shrink much per se. But because the testes shrank a lot (to about 1/3 of their original size, clearly smaller than the bunched up vas deferens), there is no longer enough weight or volume to keep the scrotal sac stretched / hang down any more. But the skin is still there, but it does seem to generally bunch up nearer to the body.
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SplitDik (imported)
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Re: Penis shrinkage due to zero testosterone
Penis shrinkage due to low testosterone is a medically proven phenomenon. I've seen some papers saying that you lose 20% of the smooth muscle. I couldn't find that, but a quick search turned up the following showing that a number of things occur to change structure and function of the penis:
http://www.doctorpg.org/health-testosterone-gel.html
There is a large body of scientific evidence which shows that androgens are important for maintaining the function and structure of penile nerves, blood vessels, muscle, connective tissue matrix, and the tunica albuginea.
Androgens are known to act on nerves which control erectile function. Studies on castrated animals have shown deterioration of cavernosal and dorsal nerves which was reversed by testosterone replacement restoring erectile capacity.
Low levels of androgens are also known to damage the lining of blood vessels in the penis inhibiting their ability to produce nitric oxide (NO) which is a substance which plays an important role in producing erections. The erectile dysfunction drug Viagra works by increasing NO levels.
Similar studies have also shown that androgens maintain penile smooth muscle content. Loss of smooth muscle due to androgen deprivation caused lower intracavernosal pressure, in other words lower blood pressure inside the penis and weaker softer erections. Similar changes to penile muscle have been seen in humans suffering from erectile dysfunction with the severity of their symptoms correlating with the degree of muscle loss. Worryingly the smooth muscle in the corpora cavernosa (the vascular inner part of the penis that fills with blood during erection) seems to be replaced by connective tissue which leads to fibrosis. This would prevent the normal expansion of the penis during erection leading to a diminished erect penis size.
A similar process occurs in the tunica albuginea which is an elastic outer layer surrounding the corpora cavernosa. This layer is normally rich in elastic fibres which allow it to expand easily when the inner corpora cavernosa blows up like a balloon as it becomes engorged with blood during erection. However in castrated animals the elastic fibres have been seen to be replaced with inelastic collagen. So again the penis is unable to expand normally.
http://www.doctorpg.org/health-testosterone-gel.html
There is a large body of scientific evidence which shows that androgens are important for maintaining the function and structure of penile nerves, blood vessels, muscle, connective tissue matrix, and the tunica albuginea.
Androgens are known to act on nerves which control erectile function. Studies on castrated animals have shown deterioration of cavernosal and dorsal nerves which was reversed by testosterone replacement restoring erectile capacity.
Low levels of androgens are also known to damage the lining of blood vessels in the penis inhibiting their ability to produce nitric oxide (NO) which is a substance which plays an important role in producing erections. The erectile dysfunction drug Viagra works by increasing NO levels.
Similar studies have also shown that androgens maintain penile smooth muscle content. Loss of smooth muscle due to androgen deprivation caused lower intracavernosal pressure, in other words lower blood pressure inside the penis and weaker softer erections. Similar changes to penile muscle have been seen in humans suffering from erectile dysfunction with the severity of their symptoms correlating with the degree of muscle loss. Worryingly the smooth muscle in the corpora cavernosa (the vascular inner part of the penis that fills with blood during erection) seems to be replaced by connective tissue which leads to fibrosis. This would prevent the normal expansion of the penis during erection leading to a diminished erect penis size.
A similar process occurs in the tunica albuginea which is an elastic outer layer surrounding the corpora cavernosa. This layer is normally rich in elastic fibres which allow it to expand easily when the inner corpora cavernosa blows up like a balloon as it becomes engorged with blood during erection. However in castrated animals the elastic fibres have been seen to be replaced with inelastic collagen. So again the penis is unable to expand normally.
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Hash (imported)
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Re: Penis shrinkage due to zero testosterone
I believe most, if not all, transsexuals use chemical castration drugs for months or years before transgender surgery. Use of these chemical castration drugs, shrink their penises. If you look at Dr. McGinn's website, you'll easily see that in some of the pre-op pictures, that most of the penises shrank dramatically before surgery. Some in the transsexual community will state that this shrinkage is not beneficial because it doesn't give the surgeon much to work with, but others will tell you that penile skin, which is used to line the new vagina, stretches enough so that it's not usually much of a problem. Scroll down and take a looksy: http://www.drchristinemcginn.com/servic ... plasty.php
Some of the penises look normal and some have obviously shrank.
Some of the penises look normal and some have obviously shrank.